anti il 6 neutralizing antibody af506 (R&D Systems)
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Anti Il 6 Neutralizing Antibody Af506, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 86 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+rat+il+6+antibody/Rat+IL-6+Antibody/pm41290054-602-27-31
Average 94 stars, based on 86 article reviews
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Recombinant:Article Title: Inhibition of HMGB1 reduces rat spinal cord astrocytic swelling and AQP4 expression after oxygen-glucose deprivation and reoxygenation via TLR4 and NF-κB signaling in an IL-6-dependent manner Article Snippet: .. Special chemicals include ethyl pyruvate (EP, Sigma-Aldrich Co., Cat# E47808, Saint Louis, MO, USA), CLI-095 (Invivo Gen, Cat# tlrl-cli95, San Diego, CA, USA), C34 (TOCRIS, Cat# 5373, Bristol, UK), recombinant HMGB1 (rHMGB1, ProSpec, Cat# pro-581-b, Rehovot, Israel), BAY 11-7082 (Sigma-Aldrich Co., Cat# B5556), IL-6 (PeproTech Inc., Cat# 400-06-2UG, Rocky Hill, NJ, USA), and neutralizing Article Title: Microglia/Macrophages and CD4 + CD25 + T Cells Enhance the Ability of Injury-Activated Lymphocytes to Reduce Traumatic Optic Neuropathy In Vitro Article Snippet: .. Monoclonal anti-rat TNF-α/TNFSF1A TNFR1/2 antibody and Incubation:Article Title: Crosstalk between interleukin-6 and corticotropin-releasing factor modulate submucosal plexus activity and colonic secretion. Article Snippet: 0889-1591/$ see front matter 2013 Elsevier Inc. A http://dx.doi.org/10.1016/j.bbi.2013.01.078 Abbreviations: CRF, corticotropin releasing facto colo-rectal distension; GI, gastrointestinal; HPA, hyp IBS, irritable bowel syndrome; IL, interleukin; IL-6R short circuit current; JAK-STAT, janus tyrosine kinas activators of transcription; MAPK, mitogen activated separation; NS, non-separated; SD, standard deviatio mean; SMP, submucosal plexus; TER, trans-epithel necrosis factor.. ⇑ Corresponding author.. Address: Department o Gateway Building, University College Cork, Cork, Irela E-mail address: d.omalley@ucc.ie (D. O’Malley). Article Title: Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism. Article Snippet: O’Malley D, Liston M, Hyland NP, Dinan TG, Cryan JF.. Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism.. Am J Physiol Gastrointest Liver Physiol 300: G241–G252, 2011. Affinity Purification:Article Title: Crosstalk between interleukin-6 and corticotropin-releasing factor modulate submucosal plexus activity and colonic secretion. Article Snippet: 0889-1591/$ see front matter 2013 Elsevier Inc. A http://dx.doi.org/10.1016/j.bbi.2013.01.078 Abbreviations: CRF, corticotropin releasing facto colo-rectal distension; GI, gastrointestinal; HPA, hyp IBS, irritable bowel syndrome; IL, interleukin; IL-6R short circuit current; JAK-STAT, janus tyrosine kinas activators of transcription; MAPK, mitogen activated separation; NS, non-separated; SD, standard deviatio mean; SMP, submucosal plexus; TER, trans-epithel necrosis factor.. ⇑ Corresponding author.. Address: Department o Gateway Building, University College Cork, Cork, Irela E-mail address: d.omalley@ucc.ie (D. O’Malley). Article Title: Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism. Article Snippet: O’Malley D, Liston M, Hyland NP, Dinan TG, Cryan JF.. Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism.. Am J Physiol Gastrointest Liver Physiol 300: G241–G252, 2011. Derivative Assay:Article Title: Crosstalk between interleukin-6 and corticotropin-releasing factor modulate submucosal plexus activity and colonic secretion. Article Snippet: 0889-1591/$ see front matter 2013 Elsevier Inc. A http://dx.doi.org/10.1016/j.bbi.2013.01.078 Abbreviations: CRF, corticotropin releasing facto colo-rectal distension; GI, gastrointestinal; HPA, hyp IBS, irritable bowel syndrome; IL, interleukin; IL-6R short circuit current; JAK-STAT, janus tyrosine kinas activators of transcription; MAPK, mitogen activated separation; NS, non-separated; SD, standard deviatio mean; SMP, submucosal plexus; TER, trans-epithel necrosis factor.. ⇑ Corresponding author.. Address: Department o Gateway Building, University College Cork, Cork, Irela E-mail address: d.omalley@ucc.ie (D. O’Malley). Neutralization:Article Title: Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism. Article Snippet: O’Malley D, Liston M, Hyland NP, Dinan TG, Cryan JF.. Colonic soluble mediators from the maternal separation model of irritable bowel syndrome activate submucosal neurons via an interleukin-6-dependent mechanism.. Am J Physiol Gastrointest Liver Physiol 300: G241–G252, 2011. Article Title: The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection. Article Snippet: Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection Xu-Hong Wei a,1, Xiao-Dong Na a,b,1, Guang-Jie Liao a, Qiu-Ying Chen a, Yu Cui a, Feng-Ying Chen c, Yong-Yong Li a, Ying Zang a,⁎, Xian-Guo Liu a,⁎ a Pain Research Center and Department of Physiology, Zhongshan Medical School of Sun Yat-Sen University, PR China b Department of Pathophysiology, Zhongshan Medical School of Sun Yat-Sen University, PR China c School of Pharmaceutical Science of Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzhou 510080, PR China ⁎ Corresponding authors at: Department of Physiology Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzho E-mail addresses: yingzang@163.com (Y. Zang), liux 1 Both authors contribute equally to this work. Saline:Article Title: Inhibition of interleukin-6 attenuates erectile dysfunction in a rat model of nerve-sparing radical prostatectomy. Article Snippet: Introduction.. The precise mechanisms underlying erectile dysfunction (ED) occurring after cavernous nerve (CN)sparing surgery remain to be determined.. Aim. Article Title: The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection. Article Snippet: Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection Xu-Hong Wei a,1, Xiao-Dong Na a,b,1, Guang-Jie Liao a, Qiu-Ying Chen a, Yu Cui a, Feng-Ying Chen c, Yong-Yong Li a, Ying Zang a,⁎, Xian-Guo Liu a,⁎ a Pain Research Center and Department of Physiology, Zhongshan Medical School of Sun Yat-Sen University, PR China b Department of Pathophysiology, Zhongshan Medical School of Sun Yat-Sen University, PR China c School of Pharmaceutical Science of Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzhou 510080, PR China ⁎ Corresponding authors at: Department of Physiology Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzho E-mail addresses: yingzang@163.com (Y. Zang), liux 1 Both authors contribute equally to this work. Control:Article Title: The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection. Article Snippet: Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection Xu-Hong Wei a,1, Xiao-Dong Na a,b,1, Guang-Jie Liao a, Qiu-Ying Chen a, Yu Cui a, Feng-Ying Chen c, Yong-Yong Li a, Ying Zang a,⁎, Xian-Guo Liu a,⁎ a Pain Research Center and Department of Physiology, Zhongshan Medical School of Sun Yat-Sen University, PR China b Department of Pathophysiology, Zhongshan Medical School of Sun Yat-Sen University, PR China c School of Pharmaceutical Science of Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzhou 510080, PR China ⁎ Corresponding authors at: Department of Physiology Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzho E-mail addresses: yingzang@163.com (Y. Zang), liux 1 Both authors contribute equally to this work. Sterility:Article Title: The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection. Article Snippet: Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection Xu-Hong Wei a,1, Xiao-Dong Na a,b,1, Guang-Jie Liao a, Qiu-Ying Chen a, Yu Cui a, Feng-Ying Chen c, Yong-Yong Li a, Ying Zang a,⁎, Xian-Guo Liu a,⁎ a Pain Research Center and Department of Physiology, Zhongshan Medical School of Sun Yat-Sen University, PR China b Department of Pathophysiology, Zhongshan Medical School of Sun Yat-Sen University, PR China c School of Pharmaceutical Science of Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzhou 510080, PR China ⁎ Corresponding authors at: Department of Physiology Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzho E-mail addresses: yingzang@163.com (Y. Zang), liux 1 Both authors contribute equally to this work. Concentration Assay:Article Title: The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection. Article Snippet: Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr The up-regulation of IL-6 in DRG and spinal dorsal horn contributes to neuropathic pain following L5 ventral root transection Xu-Hong Wei a,1, Xiao-Dong Na a,b,1, Guang-Jie Liao a, Qiu-Ying Chen a, Yu Cui a, Feng-Ying Chen c, Yong-Yong Li a, Ying Zang a,⁎, Xian-Guo Liu a,⁎ a Pain Research Center and Department of Physiology, Zhongshan Medical School of Sun Yat-Sen University, PR China b Department of Pathophysiology, Zhongshan Medical School of Sun Yat-Sen University, PR China c School of Pharmaceutical Science of Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzhou 510080, PR China ⁎ Corresponding authors at: Department of Physiology Sun Yat-Sen University, 74 Zhongshan Rd.. 2, Guangzho E-mail addresses: yingzang@163.com (Y. Zang), liux 1 Both authors contribute equally to this work. |


